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The increase in cellular calcium activates calcium-dependent proteases that lead to the proteolytic cleavage of a portion of xanthine dehydrogenase, converting it to xanthine oxidase (22)

Parallel studies indicate that physiologic dopamine signaling generally inhibits NLRP3 activity via receptor-mediated pathways, implying that the gradual loss of dopamine in PD removes an endogenous anti-inflammatory brake, exacerbating neuroinflammation

Importantly, the absence of large, controlled human studies means there is no reliable, standardized "before and after" schedule, only anecdotal reports