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Secondary plots for the rate constants of the rapid first reaction phase at variable PfGrx concentrations deviated from linearity and showed a hyperbolic correlation at millimolar GSH concentrations (Fig

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In summary, our in vitro results suggest, that TGF- and oxidative stress contribute to increased GPX3 expression in lung fibrosis in a cell-type-specific manner, upregulating GPX3 in bronchial epithelial cells and interstitial fibroblasts, respectively

The AT1-type angiotensin receptor in oxidative stress and atherogenesis