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Figure 5 4.2 Therapeutic strategies targeting lipid peroxidation and GPX4 protection Lipid peroxidation represents a central pathogenic event in ferroptosis, contributing to plasma membrane rupture and mitochondrial dysfunction

This damage was ameliorated by spermidine supplementation, which reduced inflammation, decreased antibody production and increased vascular relaxation and mitophagy (Reference Kim and Massett87)

A combination of vitamin D and L-cysteine administration significantly augmented GSH levels and lowered oxidative stress and inflammation (308, 309)

This information should be interpreted with a qualified healthcare provider to contextualize how genetic factors may relate to individual glutathione status and need